I have only seen one infectedfacet joint in my time - it is rare.
For your hypermobility patients, what is your plan of attack... i am assuming you use CMs to deal with the protective muscular spasm (which probably works via a neurophysiological effect or ischaemic pressure trying to get through all that bulk to the joint).
But once you relieve their protective strategy, they will still have the underlying hypermobility. If you don't change their stabilisation strategy, they will end up with the pain again in the same way with the same protective muscular spasm...do you have a different view/philosophy on this?