Wikipedia reference-linkFrozen shoulder is one factor that can lead to a OA shoulder... (however, it is my personal belief that the GH joint cant be classified as classic OA) I believe that all biomechanically unsound shoulders develop degenerative changes.

Before hydrodilatation, try releasing everything that attaches into the scapula (includes biceps and triceps and coracobrachialis AND pec minor) and release the pec maj and lat. It's also a good idea to perform some fascial work on the lateral border of the scapula. I have experienced an immediate increase (sometimes dramatic) in AROM of the gh joint after the first treatment. I use mainly trigger point therapy and a modified NISA approach. Treatment works on the elderly and the young and is moderately painful when subscap, teres, coracobrachialis and bicep tendons are treated. I see patients once a week when chronic for 2-3 months and each session requires 30-60 minutes of hands on. Strengthening exercises are given after the first 2-3 weeks of Tx. When acute I just do pain management because treatment forces the brain to splint the joint more aggressively. Outcomes are always positive as long as there is no calcification of the tendons. If calcification is present, AROM cannot be restored to levels prior to symptom onset but AROM can be improved so ADL's are painfree. Time of onset to time of treatment is a factor in determining if full AROM can be achieved (obviously).

Adamo