Alophysio and Damien ( let me know if you have a Phd and I'd be only too glad to add the honorific on later posts)

"the primary problem is from "lumbar protective responses" causing alteration of function and subsequent hip issues - would i be accurate with that Ginger?"
Almost.
The alteration of function is of the spine ( for detail of this I will direct the reader here The physiology of spinal pain. A theoretical model ), where an innate protective behaviour reduces movement by inducing higher paravertebral tone. This often leads to spondylitic irritations to nerve roots, which give rise to altered sensations, pain and altered patterns of recruitment of muscle. Referred events also include the effects brought about by induced changes to parasympathetic neurology, including puffiness, local colour changes, altered circulation, inflammation etc.
The "Hip" issues when these arise are brought about by pain patterns programmed in the brain , responding to nerve root nociceptive input, mapped out according to those nerves ( the brain does not recognise structures, only nerves).
First duty of any investigator is to pay attention to the most likely first, that being, that where no trauma had been a feature of the sequence of events leading to pain, that the spine plays a major role in the pain and other symptoms. I find this is invariably true when dealing with "bursitis', also with "tendonitis". For the most part these diagnoses require, at the very least, a thorough consideration of the spine.
By thorough i do not mean slump or neural tension testing, palpation of the complained of structure ( tells you nothing of value) or observations of periodicity of pain. The only reliable way to be able to confidently rule out spine as cause, is to follow the Cm protocol.