I see most posters here are still fascinated by structures and guided by the pathanatomical model inherent in the 'medical' way of thinking. When pain and apparent interference in normal functions occur they are always associated with a neural irritation somewhere. The logic I apply is that the most likely source of this is the neural interface commonly associated with referred pain events ( which include sympathetic nerve effects). That being the spine. The ubiquitous element in presentations where pain is a major feature is protective behaviour at the spine. As pain is a brain event it will not be possible to figure out wether this symptom is referred or not by considerations of the nature of this person's pain. referred pain is the same as not referred pain. Attempts at reproducing pain by overpressures etc will most often fail and give a false negative reading. The best approach is to assume the presence of a spinal neural component to this ( and other MSK ) problems and a priori go about the business of restoring a non protected , non irritated spinal root to nerves associated with and adjacent to, the complained of area. In most cases in my own approaches to so called 'shin splints' and other running related anterior leg pain, it becomes clear that referred symptoms dominate.