hi,
im swapnil doing bachlor of physiotherapy from india. im preparing for pce and npte.I think studing in group will help us out a lot.all those who want to join me out are being welcome.
Similar Threads:
hi,
im swapnil doing bachlor of physiotherapy from india. im preparing for pce and npte.I think studing in group will help us out a lot.all those who want to join me out are being welcome.
Similar Threads:
what exactly were you looking for? i'm thinking you have some specific questions??
jess
jess ans of ur questions are
the commonly used lung funtion testings used by physical therapists are
tests of ventilation
1.plethysmography
2.peak expiratory flow rate
3.forced expiratory flow volume in 1 sec.
exercise testing
it includes
1. treadmill testing
2.12 min walikng test
3.cycle ergometry
jess and anisha yes the application of breathing exercises on icu patients or the patients who must remain in bed for an extended period of time is a very critical task but with great care and regular testing and checkups we can apply it boldly.
i have pointed out a few breathing techniques other than the traditional breathing exercises.
1. insentive respiratory spirpmetry:
it is a form of ventillatory traning emphasizing sustained maximum inspirations. a synonymous term is sustained inspiratory maneuver, which is performed with or with out specification.
it increases the volume of air inspired.
the patient have to take three to four slow,easy breaths than place the spirometer and maximally inhale through the spirometer and hold the inspiaration for few seconds.
2. segmental breathing:
in this patient is taught to emphasize on localized areas of lungs while keeping others quit.hypoventilation occurs in certain areas of lungs because of chest wall fibrosis,pain and muscle guarding after susgery.
3. pursed lip breathing: it is thought to keep airways open by creating backpressure in airways.
4. glossopharyngeal breathing: it is used to increase patients inspiratory capacity when there is severe weakness of inspiratory muscles.
anisha answers for you.
1. as what i recovered from some written materials it has shown that cystic fibrosis is found only in white skinned community as europeans. so it is likly to happen that indians are not the sufferers of this condition.
2. contraindications for treadmill testing are:
1. systolic pressure should not exceed 220 to 240 mmhg.
2. diastolic pressure should not exceed 120mmhg.
3. respiration should not be labored.
4. the patient should not have perception of shortness of breath,
5. the increase in blood flow while exercing.
6. results in change in skin ,cheeks etc. they become moist warm pink
3.cardiac conditioning exercising are aerobic exercise training(conditioning):
it is an augmentation of energy utilization of the muscles by means of exercise program.
>training is dependent on exercise of sufficient intensity,duration and frequency.
>training produces a cardiovascular and or muscular adaptation and is reflected in an individuals endurance.
>training for particular sport or event is dependent on the specificity principle.
hi meenakshi nice to see you again, ill add to you in my group when you will regularly reply to us, and will regular in group studies.
hi friends the topics from my side are,
1. chronic bronchitis
2. Atelectasis
3. Nuclear imaging techniques for chest diagnosis.
hi swapnil i am happy that you are working hard to make your group members regular in their studies. that is what the real work of a good leader is.
a salute to you buddy:hat
i promise ill cooperate you and will be regular from now
anisha my questions for you as follows
1. differentiate between emphysema and chronic bronchytis?2. can the dilated alvioli be regenerated again?
3. does smoking direcly relates with bhronchytis?
thanks for the answers above swapnil
just one question- can you explain MET's and how you'd use it to determine an exercise programme for an individual?...please,
jess
hey guys.....just some research i found regarding physiotherapy and CF, which id like to share with you and also something to think about!
Thought id put it in bullet points, hence easier to read.
CF research
*antibiotic-mediated reduction in sputum bacterial density improves lung function more than bronchodilators and physio.
*conventional physio is labour-intensive, time consuming, expensive and not frequently available. instead, hidh frequency chest compression (hfcc) usues an inflatable vest linked to an air-pulsed delivery system which can be used as an alternative. it is equally safe when used in acute pulmonary axaccerbations in CF patients.
*....although there is little immediate functional improvement when conventional physio is used on a regualr basis, a 3 week period without conventional methods leads to a worsening of functional status.
*vigorous cough sessions are better than postural drainage and manual techniques alone.
*aerobic training and upper body strength training has shown to increase physical work capacity for children with CF.
...ponder away.....
jess
topics include:
valve stenosis and regurgitation
ARDS
respiratory failure
jess
Hi Ms. Handa
I just went thru ur study group and m interested in joining.
I hope im not too late.
Can u pls let me know how to proceed..
regards
sunil turlapati
hi myself chaitanya frm lucknow but studying final yr in PT.
may i know abt u ?
urrr ok guys....hope this isnt boring the hell outa you since im nt getting any quesyions you all must be awesome physios!!! 8:smokin
ok topics for you include:
1. PVD
2. INPATIENT CARDIAC REHAB
3. OUTPATIENT EXERCISE PRESCRIPTION
oh and also i've been meaning to ask u guys one thing.
i was wondering if someone could shed some BRIGHT light on these key points for respiratory patients:
respiratory acidosis
respiratory alkalosis
metabolic acidosis
metabolic alkalosis
....as well as the compensating mechanisms!
(this bit ALWAYS does my head in!)
suggestions welcome
jess
Jess
Are you studing all that stuff for the practical exam?!!!
nope the written...why?
jess
Hi,
I m a Bach holder in Physio. I want to join the group. Can you let me know the topics ur preparing at this time. I would lik to give PCE and NPTE exams.
Is there any limt on the no. of attempts for the above exams.
when is the gropu coming online for chat.....
Thank You,
regards
sunil turlapati
for jess
jess you asked for met. i hope im not confused with abrreviation if so than re reply.
MET :stands for a metabolic equivalent. a met is defined as the oygen consumed(9mili liters) per kilograms of body weight per minute(ml/kg).it is equal to approx 3.5ml/kg per minute square.
ARDS: it stands for acute respiratory distress syndrome. it is one of the leading pathologies of respiratory system.
a brief overview to ards is
it can be caused by peumonia,sepsis,smoking,aspiration,major trauma burns.
inflamation is caused at alveolar epithilium and pulmonary capillaries.pulmonary capillaries become more permeable allowing plasma and blood to leak into the interstitial and alveolar spaces, while at same time the capillaries get blocked with cellular debris and fibres. the lungs become heavy,stiff and water logged and alvioli collapse.this leads to ventillation/perfusion mismatch and hypoxaemia and patients normally require mechanical ventilatory support to achieve adequate gas exchange.
symtoms usually develop with in 24 to 48 hrs after the origianl injury or illness,but can develop 5-10 days later.
CAN IN THIS WHOLE GROUP GIVE PHYSIO MANAGEMENT OF ARDS
VALVE STENOSIS:
SIGNS:EJECTION systolic murmer
slow rising carotid pulse,redused pulse pressure
left ventricular hypertrophy,thrusting left ventricle
signs of left ventricular failure
symtoms:
exertional dysnoea
angina
pulmonary syncope
sudden death
possible investigations:
ECG(LEFT VENTRCULAR HYPERTROPHY LBBB)
CHEST RADIOGRAPH
DOPPLER
ECHO
MANAGEMENT include aortic baloon valvoplsty but of no long term value for elderly patients
for jess
mitral regurgitation:
it causes gradual dilation of the left atrium with little increase in pressure and therefore relativly few symptoms
causes:
mitral vave prolapse
dilatation of the mitral valve ring
damage to valve cusps and chordae
damage to papillary muscle.
myocardial infarction
respiratory failure:
it is broadly defined as an inability of the respiratory system to maintain blood gas valves with in normal ranges.these are of two types:
type I(hypoxaemic respiratory failure)
a decreased pao2(hypoxalmia) with a normal or low paco2 due to inadequate gas exchange.causes induced pneumonia,emphysema,fibrosing.alveolitis,sever asthma and adult respiratory distress syndrome.
typeII(ventilatory failure)
a decreased pao2 with an increased paco2(hypercapnia) caused by hyperventilation. causes include neuromuscular disorders like muscular dystrophy etc. drug related respiratory drive depressions an increase to chest wall
questions for anisha's topics
1.compare atelectesis of entire lung,right upperlobe,right middle lobe and left upper lobe with each other.
2.discuss radiographic changes seen during lung or lobar collapse.
3.about nuclear imaging tech no queries from my side anisha please difine some by your self
meenakshi smoking is one of the leading factor of major respiratory disorders,
in case of chronic bhronchytis or emphysema also it plays a major role. what was you really wanted to ask with your question?
for physiopati,chaitanyamohansuri,sunnyphysio
if you want to join the group join the current topic and reply regularly
u will get password after some time for password oriented room
wow i was begining to think everyone had vanished!....thanks for the detailed replies however the ards, valve regurgitation and resp failure stuff you kindly added, was actually th topics that i'd prepared. if you were simply adding more info- then thats great. but i hope you didnt think that i'd asked all those questions!!!! much appreciate the METs info. (the question which i asked help with)...all is well knowing what it is...but how can 'we' use it in a clinical setting??
jess